Documented study links electromagnetic hypersensitivity as a real-time health issue that actually can be verified using standard medical procedures and testing capabilities. An international group of researchers aced it when they published their findings from the clinical study “Metabolic and Genetic Screening of Electromagnetic Hypersensitive Subjects as a Feasible Tool for Diagnostics and Intervention” in the November 2014 issue of Mediators of Inflammation. Please click on the link to read the entire study.

[ed. extracted from the study] In this study, the working hypothesis was that EHS, as previously proposed for MCS and other environmental SRI [19, 22], may as well be based on aberrant responses to physic or chemical xenobiotic stressors through airborne or other routes of exposure, due to inherited or/and acquired dysfunction of the chemical defensive system, that is the interrelated network of phase I and II xenobiotic-metabolizing and antioxidant enzymes [19]. Based on the results of our past clinical studies on MCS, FM, and CFS, we sought to assess if similar profiles of metabolic or genetic dysfunctions could be found in those subjects self-reporting EHS phenotype. To this purpose, we measured possible alterations of a previously identified panel of twelve blood redox and lipid parameters and frequencies of selected genetic mutated variants of a set of drug-metabolizing enzymes and transcription factors with first-line roles in the detoxification of physical and chemical xenobiotics, in a group of 153 patients self-reporting EHS symptoms, co-morbid in most cases with different degrees of MCS symptoms. Results were compared to those obtained on 147 MCS patients without EHS symptoms and on a healthy control group of 132 age- and sex-matched subjects, all groups enrolled within the Italian population.

Among EMFs emissions recognized as trigger factors in the group of 153 patients self-reporting electromagnetic hypersensitivity-EHS, video display units and television were the most frequently reported sources (75% of patients), followed by mobile and landline phones (53%) and by domestic appliances (48%). Further developments must necessarily include a more objective and standardized classification of individual electromagnetic sensitivity scores, to conclusively assess the proposed parameters as a distinctive and specific panel of disease biomarkers for EHS. Our findings will hopefully contribute, in combination with the so-far putative genetic-risk factors, a better molecular definition of environmental-borne sensitivity-related illnesses and a tool to discriminate single SRI comorbidities, based on sufficiently proven molecular evidences able to gain clinical consensus.

See the website www.SAFEhelpsyou.org for more information on developing projects for referrals to areas that are safe for persons with EHS andMCS.